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Regulation of autophagy by metabolic and stress signaling pathways in the heart
Regulation of autophagy by metabolic and stress signaling pathways in the heart
Bnip3-mediated mitochondrial autophagy is independent of the mitochondrial permeability transition pore
Bnip3-mediated mitochondrial autophagy is independent of the mitochondrial permeability transition pore
Loss of MCL-1 leads to impaired autophagy and rapid development of heart failure
Loss of MCL-1 leads to impaired autophagy and rapid development of heart failure
Mitochondrial autophagy by Bnip3 involves Drp1-mediated mitochondrial fission and recruitment of Parkin in cardiac myocytes
Mitochondrial autophagy by Bnip3 involves Drp1-mediated mitochondrial fission and recruitment of Parkin in cardiac myocytes
Cellular redox status determines sensitivity to BNIP3-mediated cell death in cardiac myocytes
Cellular redox status determines sensitivity to BNIP3-mediated cell death in cardiac myocytes