Search results

(1 - 5 of 5)
Label
Regulation of autophagy by metabolic and stress signaling pathways in the heart
Youngil, L., Lee, H.-Y., & Gustafsson, A. B. (2012). Regulation of autophagy by metabolic and stress signaling pathways in the heart. Journal of Cardiovascular Pharmacology, 60(2), 118-124. doi:10.1097/FJC.0b013e318256cdd0
Bnip3-mediated mitochondrial autophagy is independent of the mitochondrial permeability transition pore
Quinsay, M. N., Thomas, R. L., Lee, Y., & Gustafsson, A. B. (2010). Bnip3-mediated mitochondrial autophagy is independent of the mitochondrial permeability transition pore. Autophagy, 6(7), 855-862. doi:10.4161/auto.6.7.13005.
Loss of MCL-1 leads to impaired autophagy and rapid development of heart failure
Thomas, R. L., Roberts, D. J., Kubli, D. A., Lee, Y., Quinsay, M. N., Owens, J. B., Fischer, K. M., Sussman, M. A., Miyamoto, S., & Gustafsson, Åsa B. (2013). Loss of MCL-1 leads to impaired autophagy and rapid development of heart failure. Genes & Development, 27(12), 1365-1377. doi:10.1101/gad.215871.113
Mitochondrial autophagy by Bnip3 involves Drp1-mediated mitochondrial fission and recruitment of Parkin in cardiac myocytes
Lee, Y., Lee, H.-Y., Hanna, R. A., & Gustafsson, A. B. (2011). Mitochondrial autophagy by Bnip3 involves Drp1-mediated mitochondrial fission and recruitment of Parkin in cardiac myocytes. American Journal of Physiology. Heart and Circulatory Physiology (Print), 301(5), H1924-H1931. doi:10.1152/ajpheart.00368.2011
Cellular redox status determines sensitivity to BNIP3-mediated cell death in cardiac myocytes
Lee, Y., Kubli, D. A., Hanna, R. A., Cortez, M. Q., Lee, H.-Y., Miyamoto, S., & Gustafsson, A. B. (2015). Cellular redox status determines sensitivity to BNIP3-mediated cell death in cardiac myocytes. American Journal of Physiology. Cell Physiology, 308, C983-C992. doi:10.1152/ajpcell.00273.2014