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Bnip3-mediated mitochondrial autophagy is independent of the mitochondrial permeability transition pore
Bnip3-mediated mitochondrial autophagy is independent of the mitochondrial permeability transition pore
Cellular redox status determines sensitivity to BNIP3-mediated cell death in cardiac myocytes
Cellular redox status determines sensitivity to BNIP3-mediated cell death in cardiac myocytes
Loss of MCL-1 leads to impaired autophagy and rapid development of heart failure
Loss of MCL-1 leads to impaired autophagy and rapid development of heart failure
Mitochondrial antioxidant enzymes and endurance exercise-induced cardioprotection against ischemia-reperfusion injury
Mitochondrial antioxidant enzymes and endurance exercise-induced cardioprotection against ischemia-reperfusion injury
Mitochondrial autophagy by Bnip3 involves Drp1-mediated mitochondrial fission and recruitment of Parkin in cardiac myocytes
Mitochondrial autophagy by Bnip3 involves Drp1-mediated mitochondrial fission and recruitment of Parkin in cardiac myocytes
Regulation of autophagy by metabolic and stress signaling pathways in the heart
Regulation of autophagy by metabolic and stress signaling pathways in the heart
Fast twitch skeletal muscle remodeling by prolonged endurance exercise is associated with crosstalk between anabolic and catabolic signaling pathways in mice
Fast twitch skeletal muscle remodeling by prolonged endurance exercise is associated with crosstalk between anabolic and catabolic signaling pathways in mice